Cause

Injury Recovery Support

Print­er Friend­ly PDF

Fast Track:

Overview

Injury recov­ery is always rate lim­it­ed by some­thing. The ques­tion is what…

This How­To pro­vides a gen­er­al guide­line to opti­mize heal­ing for injuries with pri­ma­ry focus on joint and con­nec­tive tis­sue. We assert there are three cofac­tors in injury in any heal­ing rate:

Cells know how to put them­selves back togeth­er. The chal­lenge is to opti­mize the envi­ron­ment so they can do it as quick­ly as pos­si­ble.

Rapid heal­ing is a sur­vival advan­tage — and is not fun­da­men­tal­ly lim­it­ed by the organ­ism, but by the effi­cien­cy of the organ­ism repair­ing itself.

Injury Basics

Sev­er­al fac­tors that gov­ern the injury process:

  1. Ini­tial trau­ma dam­ages tis­sues;
  2. Inflam­ma­to­ry process “clos­es off” the injury area (and sac­ri­fices near­by tis­sue);
  3. Recon­struc­tion restores the area to nor­mal.

Accel­er­at­ed recov­ery ther­a­py involves three prin­ci­ples:

  • Man­age inflam­ma­tion to min­i­mize sec­ondary dam­age;
  • Opti­mize nutri­ents includ­ing oxy­gen dur­ing recov­ery;
  • Opti­mize waste prod­uct flow out of the injury area.

Modulated Inflammation

Sec­ondary dam­age that devel­ops after the ini­tial trau­ma, usu­al­ly rep­re­sents the bulk of tis­sue. Min­i­miz­ing the “sec­ondary dam­age” by con­trol­ling ini­tial inflam­ma­tion lim­its the amount of heal­ing required. Any­thing which reduces sec­ondary dam­age reduces heal­ing time because it makes for less heal­ing.

An ini­tial trau­ma caus­es plas­ma and lym­pho­cytes to flow to an area. This move­ment brings the both nutri­ents and oxy­gen to sup­port the heal­ing process.

Plas­ma oxy­gen sat­u­ra­tion has two influ­ences:

  • Oxy­gen and nutri­ent rich plas­ma enables more tis­sue near the injury to sur­vive while deplet­ed plas­ma per­mits more local dam­age. Oxygen/nutrient avail­abil­i­ty is inverse­ly pro­por­tion­al to inflam­ma­to­ry tis­sue dam­age;
  • Heal­ing effi­cien­cy for non-vas­cu­lar tis­sue is pro­por­tion­al to the nutri­ents and oxy­gen avail­able in the plas­ma;
  • Post trau­mat­ic Oxygen/Nutrient avail­abil­i­ty results from plas­ma con­cen­tra­tion and mobil­i­ty of the plas­ma.

These fac­tors con­trol the heal­ing speed. Indi­vid­u­als with high plas­ma oxy­gen lev­els and suc­cess­ful diges­tion heal much faster, up to 10x, than expect­ed:

  1. Less sec­ondary dam­age from inflam­ma­tion;
  2. Con­nec­tive tis­sue has more ener­gy, oxy­gen and nutri­ents to rebuild itself.

Diffusion Nutrient Pathways

Con­nec­tive tis­sue does not have cap­il­lar­ies which bring blood close to con­nec­tive tis­sue. Nutri­ents and oxy­gen must reach these tis­sues by oth­er mech­a­nisms.

Note the bulk of the con­nec­tive tis­sues. The effi­cien­cy of dif­fu­sion mech­a­nisms con­trol nutri­ent and oxy­gen avail­abil­i­ty to con­nec­tive tis­sue.

From a ther­a­py view, restora­tion of dif­fu­sion path­way per­for­mance accel­er­ates heal­ing of non-vas­cu­lar­ized tis­sue. The Mod­el:

  1. Lim­it dam­age with inflam­ma­tion man­age­ment,
  2. Restore vas­cu­lar per­for­mance;
  3. Sup­port Cleanup;
  4. Opti­mize Sat­u­rate Nutri­ent Mobil­i­ty
  5. Sup­ply sys­temic Sat­u­rate Nutri­ents;
  6. Max­i­mize Tis­sue Oxy­gen to injury with sat­u­rate path­ways.
Please vis­it the care dis­cus­sion.

      
      
    

Leave a Reply

Your email address will not be published.

Injury Recovery