Priority Summary

The data presents several prioritiy candidates. The table suggests protocol priorities with a 1-5 grade.

Strategy Notes

At this time digestive mineralization and liver toxicity present the largest barriers to metabolic performance and healing. Urinary rH2 numbers suggest that digestive insufficiency may be caused by chlorine deficiency, likely helped with large quantities of Betaine during meals.

Urinary surface tension may be elevated due to cellular shutdown limiting exchange. The normal approach is to add 100K-1M units of Emulsified Vitamin A, combined with magnesium thiosulfate, which often helps to open cells. This is a low priority because of long toxic history. Toxicity likely suggests that cells may be closed to avoid toxin absorption, so primary focus should remain on lymph and systemic detoxification.

Decreased glucose suggests glucose metabolism and potential weakness in the tricarb pathway. It could also be driven by excessive bacteria, indicated in blood survey, overconsuming glucose. Likely

Metabolic Assessment

The absence of acute fatigue complaint, and absence of sleep, suggest that symptoms are most likely related to catabolic biased metabolism. Likewise a semi normal UpH, suggestive of catabolic metabolism, suggests that anabolic bias protocol is not sufficiently apparent to merit action.

Anabolic versus Catabolic Discussion: 2:43

Vitamin C levels suggest acute oxidative stress. Glutathione regeneration in the liver is likely deficient. This suggests acute systemic oxidative stress as indicated by parallel elevated rH2 levels in saliva and urine and supported by urinary and salivary NH4 levels.

Conversely, the long term absence of sleep suggests catabolic metabolism. Supplement to support sleep is appropriate.

Supplemental

• Chemical Sensitivity Survey
• Alliance Membership