Return to Care

Mechanism

Abstract

This pre­sen­ta­tion seg­ment describes the series of effects that lead to the choke mech­a­nism. Basic top­ics:

  • Cel­lu­lar stress event, Trig­gers Na/K imbal­ance, Inflates the cell, lim­its Future Flow, plas­ma only, forces anaer­o­bic, and locks stress.

The Vascular Network

The human vas­cu­lar sys­tem is a net­work of about 50,000 miles of tubes that car­ry blood. This blood con­tin­u­ous­ly deliv­ers oxy­gen and nutri­ents to body’s cells.

The health of vas­cu­lar net­work is crit­i­cal. Any vas­cu­lar fail­ure that shuts off blood to cells, suf­fo­cates down­stream cells — usu­al­ly with­in moments.

Vas­cu­lar con­di­tions that lim­it blood flow, reduces the blood sup­ply, trig­ger cel­lu­lar dis­tress and self preser­va­tion.

Fac­tors that affect vas­cu­lar heath are impor­tant to the entire body.

Vascular Oxygen Supply

The vas­cu­lar sys­tem is made of cells that also require nutri­ents and oxy­gen. Vas­cu­lar cells expe­ri­ence blood like a bystander near an inter­state and watch Oxy­gen rich red blood cells zoom by — loaded with oxy­gen.

But blood veloc­i­ty, and CO2 swap mech­a­nisms pre­vent vas­cu­lar cells from access­ing RBC bound oxy­gen.

This means that vas­cu­lar cells have live on the oxy­gen from some­place else. They must use the oxy­gen dis­solved in bulk flu­id, or plas­ma.

Plasma Oxygen

Through­out med­ical his­to­ry, plas­ma oxy­gen has been a curios­i­ty. Med­ical texts rec­og­nize a nor­mal con­cen­tra­tion at about 3 ml/liter, tiny com­pared to that the quan­ti­ty bound to red blood cells.

But tiny does not mean unim­por­tant — this seem­ing­ly small quan­ti­ty of oxy­gen keeps the vas­cu­lar sys­tem alive.

Until 1987, every­body except Man­fred von Ardenne, over­looked the impor­tance of this oxy­gen.

Even now, it is not con­sid­ered a fac­tor in health or dis­ease even though noth­ing could be fur­ther from the truth.

Plasma Hypoxia

Plas­ma hypox­ia is insuf­fi­cient oxy­gen in blood plas­ma to main­tain vas­cu­lar cell metab­o­lism.

Con­di­tions that reduce plas­ma oxy­gen below a crit­i­cal lev­el, can dam­age the vas­cu­lar sys­tem, like the absence of oxy­gen dam­ages any cell.

One of the hid­den effects of stress is res­pi­ra­to­ry com­pen­sa­tion which decreas­es the amount of oxy­gen dis­solved in plas­ma.

Flow And Concentration

There are two known fac­tors that gov­ern usable oxy­gen avail­able to vas­cu­lar cells, flow and con­cen­tra­tion.

Flow is the vol­ume of plas­ma that comes in con­tact with vas­cu­lar cells while con­cen­tra­tion is the total amount of dis­solved oxy­gen.

Concentration Variables

Increas­es in res­pi­ra­to­ry tur­bu­lence, like breath­ing hard, increase sol­u­ble oxy­gen lev­els; while shal­low breath, reduce lev­els.

Dis­solved solids, like salt, and meta­bol­ic waste, reduce oxy­gen sol­u­bil­i­ty. Sea water holds 25% less oxy­gen than fresh water, and blood plas­ma holds 43% less oxy­gen than sea water.

Con­di­tions that increase plas­ma-solutes, or tox­ins, decrease plas­ma oxy­gen lev­els.

This is the hid­den con­nec­tion between blood tox­ins, and the vas­cu­lar sys­tem. Plas­ma tox­ins reduce sol­u­ble oxy­gen, and there­fore reduce vital­i­ty of vas­cu­lar tis­sue.

Flow Variables

Blood is not water — it is a col­loidal sus­pen­sion gov­erned by elec­tro­sta­t­ic repul­sive forces. Sus­pend­ed par­ti­cles, like blood cells, must have enough elec­tri­al ener­gy to repel each oth­er, oth­er­wise they form clumps that clog the vas­cu­lar sys­tem.

When clumps clog nar­row­ing vas­cu­lar fun­nels, arte­ri­oles clog, flow stops, and down­stream tis­sue suf­fo­cates after oxy­gen is used up. This caus­es cel­lu­lar hypox­ia, which trans­forms sludge blood, into a destruc­tive vas­cu­lar inflam­ma­tion.

Vascular Chokes

When the vas­cu­lar inner skin, or endothe­li­um, becomes oxy­gen deprived, ener­gy metab­o­lism fail­ure dis­rupts sodi­um potas­si­um ion exchange, and these cells accu­mu­late sodi­um, which caus­es them to bloat. Bloat­ing reduces the vas­cu­lar diam­e­ter, and lim­its flow.

The diam­e­ter of a healthy cap­il­lary is small­er than a healthy red blood cell so that blood cells must bend or fold to go through cap­il­lar­ies. Bloat­ing lim­its flow to plas­ma flu­id, and per­haps red blood cells that may shrink to avoid clog­ging the cap­il­lary.

Stuck Chokes

Chokes are a stuck effect because the oxy­gen required to requred to to release them can­not arrive because the pipe is well — choked.

Man­fred von Ardenne devel­oped a sys­tem for releas­ing these chokes, we will talk more about that lat­er.

Log­i­cal­ly, the low­est con­cen­tra­tion of oxy­gen at the very end of the pipe. In the vas­cu­lar sys­tem, this is at the venous end of the cap­il­lar­ies, just before the blood starts the return jour­ney to the heart.

Ardenne and Leowe, dis­cov­ered chokes exact­ly here, at the area of low­est oxy­gen con­cen­tra­tion. The chokes were small enough to lim­it blood flow, but big enough to allow enough plas­ma to pass.

This was the mag­ic recipe for stuck. It was enough to keep the tis­sue alive, but not enough to thrive — Stuck cell stress.

Cell Distress

Body cells fed by the cap­il­lar­ies enter pro­gres­sive process of dis­tress after cap­il­lar­ies choke.

Reduced diam­e­ter forces these cells to sur­vive on plas­ma, which still fits through reduced diam­e­ter. Plas­ma car­ries glu­cose, so cells switch to anaer­o­bic or ener­gy pow­er to sur­vive.

Dis­tress pro­gress­es as body cell:

  • Ener­gy decreas­es from 19 to 1, with­out oxy­gen;
  • So per­for­mance decreas­es by the same;
  • Anaer­o­bic cells stop pro­duc­ing car­bon diox­ide
  • So cap­il­lar­ies valves remain shut;
  • Anaer­o­bic cells pro­duce acids;
  • so acids accu­mu­late in the region,
  • and the area becomes per­ma­nent­ly acidic;
  • and the area remains per­ma­nent­ly dis­tressed.

Distress Accumulates

These events cre­ates a local weak­ness, any­where, and even­tu­al­ly every­where in the body when they pile up.

The role and loca­tion of each dis­tressed area deter­mines effect. Cell weak­ness erodes per­for­mance and acidic waste caus­es irri­ta­tion, and local pH imbal­ance.

As more of the body enters dis­tress, vital­i­ty decreas­es, and health issues emerge.

Dis­tressed tis­sue:

  • In mus­cle is sore and weak;
  • In the brain caus­es motor or cog­ni­tive dys­func­tion;
  • In immune sys­tem incras­es infec­tion vul­ner­a­bil­i­ty;
  • and so on.

Large amounts of dis­tressed tis­sue cre­ate body-wide effects which inter­fere with glu­cose, pH and oth­er reg­u­la­to­ry sys­tems. We will address these effects lat­er.

Long Term Distress

These dis­tress effects from cap­il­lary chokes tend to be per­ma­nent because the meta­bol­ic cir­cum­stances which reverse them rarely occur.

Remem­ber plas­ma oxy­gen we talked about ear­li­er?

In the next sec­tion we will explain how to use it to release tis­sue dis­tress.

References

http://www2.estrellamountain.edu/faculty/farabee/biobk/BioBookcircSYS.html

http://faculty.stcc.edu/AandP/AP/imagesAP2/bloodvessels/capreal.gif

http://www.capillarycirculation.com/CapillaryEchange.html

More Information

The fol­low­ing parts of this pre­sen­ta­tion pro­vide more detailed infor­ma­tion regard­ing phys­i­ol­o­gy, his­to­ry and much more.

We have ded­i­cat­ed an entire web site to these sys­tems. Go to whnlive.com. Hov­er over the Tech­nol­o­gy Menu, and click the Alti­tude Con­trast menu item to access this site.


      
      
    

Leave a Reply

Your email address will not be published.

Fatigue